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Known causes of secondary hypertension include sleep apnea, chronic kidney disease, primary aldosteronism, renovascular disease, chronic steroid therapy, Cushing's syndrome, pheochromocytoma, coarctation of the aorta, and thyroid or parathyroid disease.

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Without the detection and correction of a reversible cause or lesion, the long-term prognosis of patients who present with acute cardiogenic pulmonary edema is poor.

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In general, inhalation facilitates muscle activity and is therefore useful during the isometric phase, while exhalation promotes inhibition and therefore helps relaxation.

In addition, the relationship between left ventricular fractional shortening change over time and hazard (nonparametric approach) displays an interaction with treatment group with crossing of the curves at a point close to the zero value for such a change (Fig 6).

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At the end of the surgery, the residual neuromuscular blockade was reversed with a standard dose of neostigmine and atropine.

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With beta blockers, the more precise it targets the Beta1 receptor the less side effects (in theory).

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Because of its beta 1-selectivity, this is less likely with Bisoprolol fumarate.

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The side effects featured here are based on those most frequently appearing in user posts on the Internet.

ACE inhibitors may be used together with diuretics.

Bisoprolol has high selectivity for beta-1 ARs with a beta-1 to beta-2 antagonist activity ratio of 119.

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It appears from our findings that the most important goal of therapy in heart failure where prognosis is concerned is more the preservation of left ventricular function and prevention of deterioration than the achievement of the greatest left ventricular function improvement.

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With severe heart failure, doctors sometimes recommend the use of this device to provide oxygen to the body.

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In infants and children, metabolic and genetic familial causes of congestive cardiopathy, such as carnitine and selenium deficiency, are more common than in adults.

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Many BB are metabolized by the liver through the CYP pathway, in particular via the CYP2D6 isoform; in humans, the CYP2D6 enzyme is highly polymorphic and this characteristic has been often referred to explain the inter-individual variability observed in the plasma levels of drugs such as carvedilol and in the responses to treatment with BB in HF.

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Sudden death is the most prevalent cause of death in the early course of CHF and in mildly symptomatic CHF, and although there is limited evidence that ACE inhibitors prevent sudden death in CHF patients, β-blockers are well documented in this regard.

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